訪問者：首都大学東京 Dilina Tuerde
【研修報告】Neuroscience 2018, 5th RNA Metabolism in Neurological Disease Conference (Society for Neuroscience)
I am very delighted that I had the opportunity to attend the 48th society for neuroscience in San Diego, 3-7 November, 2018. The conference was a very well organized and all around excellent five days of learning for me. There were more than thirteen thousand oral and poster presentations happening at a given time and specializing in research across a spectrum from a cellular level all the way to cognitive systems. It provided high- quality opportunity for us to share, learn and discuss novel and important findings in the field of neuroscience.
My submitted abstract entitled “Tau phosphorylation at AT8 pathological site during brain development”, was accepted for an oral presentation in nanosymposium on 4th Nov. During my presentation, the mostly exciting thing is that my research gained Prof. Eckhard Mandelkow`s interest who is an outstanding scientist in the field of Alzheimer`s disease and a principal investigator in the German Center for Neurodegenerative Diseases in Bonn (DZNE). He asked me several questions and also provided me with precious suggestions. It was very exciting and meaningful time for me. It is my honor to have the opportunity to discuss research with him face-to-face and get his valuable guidance. This would be an unforgettable memory for me and also boost my confidence and passion to do Alzheimer`s disease research.
There were a lot of excellent Alzheimer`s disease minisymposium or nanosymposium given by extremely prominent neuroscientists. One of the mostly wonderful symposium for me that mechanisms of tau oligomer-induced synaptic impairment and potential treatment strategies. It was interestingly discussed that supporting a role for tau oligomers in disease initiation and progression and explore therapeutic strategies for inhibiting formation of tau oligomers. From these great number of new findings, I felt rapid progress is being made in understanding Alzheimer`s disease and cognitive deficits at the genetic, circuit and molecular level. In addition to attending symposiums, I spent my time strolling around and reading many wonderful posters while also discussing with several brilliant researchers.
From this experience of participation, I felt society for neuroscience lets us know if we are heading in the right direction by share our results with others and makes us stronger in the profession by receive valuable feedback that might contribute to our future research directions. It was indeed an excellent occasion for me to develop ideas, raise visibility, and get inspired. I greatly thankful to Brain Protein Aging and Dementia Control project for supporting my travel to attend SFN and gain those wonderful and precious experiences.
My current study focused on the physiological function of AT8 tau in the developing brain and how is the mechanism of physiological phosphorylation of AT8 site related to AD development. Discussions with prominent professors and attending excellent presentations exposed me to interesting experimental design and many novel techniques which will greatly improve quality of current research in future. It also provided me an opportunity to establish a network of connections and increased the chance for getting new collaborators for current research.
Tokyo Metropolitan University
Department of Biological Sciences